• Korean Journal of Plant Taxonomy
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• v.45 no.2
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• pp.109-113
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• 2015

Fieldwork in Lao PDR during 2007-2008 resulted in the discovery of 15 unrecorded fern species among the flora of Lao PDR. All have been identified in other countries in southeastern Asia, as well as from other tropical areas. The species are as follows: Asplenicaceae (Asplenium delavayi, A. rockier), Athyriaceae (Anisocampium cuspidatum), Dennstaedtiaceae (Microlepia hookeriana, Pteridium revolutum), Dryopteridaceae (Didymochlaena truncatula, Dryopteris cochleata, Polystichum biaristatum, P. lindsaefolium), Gleicheniaceae (Dicranopteris curranii), Hypodematiaceae (Hypodematium crenatum), Lindsaeaceae (Lindsaea heterophylla), Pteridaceae (Pteris decrescens), Polypodiaceae (Pyrrosia nummularifolia) and Thelypteridaceae (Cyclosorus dentatus).

• CELLMED
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• v.2 no.4
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• pp.34.1-34.5
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• 2012

The present study aimed to determine the hepatoprotective activity of the chloroform extract of D. linearis leaves (CEDL) using the paracetamol (PCM)- and carbon tetrachloride ($CCl_4$)-induced liver injury models in rats. The rats received $dH_2O$ (negative control), 200 mg/kg of silymarin (positive control) or CEDL (50, 250 and 500 mg/kg) orally once daily for 7 days and then were subjected to the hepatotoxic induction on the $7^{th}$ day. The samples (i.e. blood and liver) were collected and underwent biochemical and microscopical analysis, respectively. From the data obtained, both inducers caused significant (p < 0.05) increase in the levels of AST and ALT when compared to the control group, which were significantly (p < 0.05) reduced by CEDL in a generally dose-dependent manner. These biochemical findings were supported by the histopathological analysis and histological scoring. In conclusion, CEDL possesses potential hepatoprotective activity, which could be associated with its flavonoid and tannin contents with the mechanisms of hepatoprotection linked to either its antioxidant or anti-inflammtory/immunomodulating activities. Further in-depth studies are required to identify the responsible bioactive compound.