• Title/Summary/Keyword: Fahr disease

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A Case Report of a Patient of Basal Ganglia Calcification Suggestive of Fahr's Disease Treated with Korean Medicinal Treatment (Fahr disease가 의심되는 기저핵 석회화 환자에 대한 한방 처치 1례)

  • Choi, Hyun-jeong;Ha, Jeong-been;Lew, Jae-hwan
    • The Journal of Internal Korean Medicine
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    • v.40 no.2
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    • pp.279-285
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    • 2019
  • Objective: This study presents the case of a 74-year-old male patient with basal ganglia calcification suggestive of Fahr's disease and the effectiveness of Korean medicinal treatment. Methods: We treated this patient with traditional Korean medicine and measured symptom severity using the Numeral Rating Scale (NRS). Results: After treatment, most pathological symptoms had decreased, and there was a gradual decline in the NRS of patient's symptoms. Conclusions: Korean medicinal treatment can be a solution for patients with basal ganglia calcification.

A Case Report of Fahr Disease with Tremor (진전을 동반한 Fahr Disease 환자 치험례)

  • Kim, Tae-Yeon;Shin, Woo-Jae;Park, Yu-Jin;Moon, Joo-Ho;Shin, Seon-Mi;Kim, Gi-Tae;Go, Heung
    • The Journal of Internal Korean Medicine
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    • v.32 no.3
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    • pp.435-443
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    • 2011
  • Fahr disease (FD) is a rare neurological disorder characterized by presence of abnormal and associated cell loss in certain areas of the brain, mostly in basal ganglia, thalamus, cerebellum and subcortical areas. Approximately two-thirds of the patients are symptomatic. The most common neurological manifestations include movement disorders, cognitive impairment, cerebellar signs and speech disorders. We report one case of estimated FD through brain computed tomography (bilateral calcifications of basal ganglia, thalamus, centrum semiovale, subcortical white matter of occipital lobes, cerebellum). At the first time of treatment, he complained of tremors in his upper limbs. We diagnosed the patient as deficiency of qi (氣) and movement of phlegm-heat-wind (痰熱風動) type according to symptoms and treated by herbs and acupuncture of oriental medicine. During treatments, we evaluated how well the oriental medical treatments were working using visual analogue scale (VAS) and amplitude of hands. After the oriental medical treatments about tremor, VAS dropped from 10 to 2 and amplitude of hands from 20 mm to 2 mm, but the ratio of brain calcifications was not changed. This study suggests that oriental medical treatments can be applicable to improve FD.

Bilateral Striopallidodentate Salcinosis on CT and MRI : Case Report (양측성 선조-담창-치상액 석회증의 전산화단층촬영과 자기공명영상 소견 : 증례보고)

  • Lee Jong Deok
    • Journal of Physiology & Pathology in Korean Medicine
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    • v.18 no.2
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    • pp.621-625
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    • 2004
  • Bilateral striopallidodentate calcinosis, popularly referred to as Fahr's disease, is a disorder radiologically characterized by bilateral calcifications of the basal ganglia, thalami, dentate nuclei of the cerebellum, and the white matter of the cerebral hemisphere without serum calcium-phosphorus metabolism and related endocrinologic abnormalities. Intracranial calcifications are easily visible as high-density on CT. On magnetic resonance images, the calcifications exhibit different signal intensities. The differences in signal intensity are thought to be related to the stage of the disease, differences in calcium metabolism, and the volume of the calcium deposit. Based on literature review, I report the case of a 63 year man with bilateral symmetrical calcification in the basal ganglia, dentate nuclei of the cerebellum, and the white matter of the cerebral hemisphere who present a 5 year history of progressive dysarthria associated with left thalamic infarction.

Familial Idiopathic Basal Ganglia Calcification

  • Shin, Dong-Ah;Gong, Tae-Sik;Shin, Dong-Gyu;Kim, Hyoung-Ihl
    • Journal of Korean Neurosurgical Society
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    • v.40 no.3
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    • pp.196-198
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    • 2006
  • Familial idiopathic basal ganglia calcification[FIBGC] is an inheritable neurological condition characterized by calcium deposits in the basal ganglia and extra-basal ganglia areas. The condition manifests as parkinsonism and other variable neuropsychiatric symptoms. FIBGC is a rare condition, and its pathophysiology has not yet been fully elucidated. Here we report the results of a clinical study of two related patients diagnosed with FIBGC.