• 제목/요약/키워드: Coracoid impingement syndrome

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상완골 소결절에 발생한 부정 유합의 관절경적 치료 - 증례 보고 - (Arthroscopic Treatment of Lesser tuberosity Malunion - A Case Report -)

  • 손훈상;정덕문;신상진
    • 대한관절경학회지
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    • 제12권3호
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    • pp.217-221
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    • 2008
  • 오구 충돌 증후군은 상완골 전상방 면과 오구견봉 궁 후외측면의 비정상적 접촉에 의해 발생하며, 그 원인으로는 외상성, 특발성 및 의인성 인자가 있다. 그 중 외상에 의한 경우는 견갑골 경부, 오구 돌기, 또는 상완골 소결절 골절이 원인이 될 수 있다. 상완골 소결절 부정 유합에 의한 오구 충돌 증후군은 드문 질환으로 치료에 대해서는 주로 개방적 수술이 사용되어 왔으나 관절경적 오구 돌기 성형술과 함께 소결절의 경피적 고정술을 보고한 예는 없었다. 이에 저자들은 상완골 소결절 부정 유합으로 발생한 오구 충돌 증후군에 대하여 관절경적 수술로 치유된 1 예를 경험하였기에 문헌 고찰과 함께 보고하고자 한다.

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견관절 충돌 증후군 환자에서 오훼 견봉궁의 자기공명 영상 평가 (Evaluation of Coraco-Acromial Arch in Patients with Impingement Syndrome)

  • 이광진;변기용;권순태;변규환
    • Clinics in Shoulder and Elbow
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    • 제2권1호
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    • pp.35-40
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    • 1999
  • Impingement syndrome is caused by a conflictual status between rotator cuff, subacromial bursa and anatomic and functional coracoacromial arch. The purpose of this study was to assessment the coracoacromial arch by MRI and to determine major factors among five components of coracoacromial arch. We analyzed forty-two cases of clinical impingement sign and test positive and postoperative confirmed diagnosed from March, 1991 to January, 1999. We evaluated acromial end abnormality according to the Bigliani acromial type and formation of osteophyte. Clavicular end abnormality classified flat, outward protrusion, inward protrusion to coracoacromial arch. Acromioclavicular joint abnormalities were advanced osteoarthritis and positive signal change. Coracoacromial ligament thickening was above 2 mm in oblique sagittal image. Coracoid process abnormality was inward protrusion to coracoacromial arch. All consecutive patients abnormalities were as follows: clavicular end osteophyte formation and inward protrusion to coracoacrmial arch were 30%, acromial end osteophyte formation was 28%, advanced acromioclavicular joint arthritis and osteophyte formation were 56%, coracoacromial ligament thickening was 24% and no coracoid process inward protrusion to coracoacromial arch. Impingement syndrome combined with rotator cuff tear group abnormalities were clavicular end(40%), acromial end(40%), acromioclavicular joint(20%), coracoacromialligament(20%) and coracoid process abnormality(0%) respectively. Only impingement syndrome group abnormalities were clavicular end(25%), acromial end(31%), acromioclavicular joint(62%), coracoacromial ligament(25%) and coracoid process(0%) respectively. Acromial type I(flat) were 6 cases, type II(curved) were 26 cases and type III(hooked) were 10 cases. We concluded that the most important contributing factors for impingement syndrome was acromial type and second was acromioclavicular joint arthritis and bony spur formation.

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양궁 선수의 오구 충돌 증후군의 치료 후 발생한 회전근 간격의 손상에 대한 치료 - 증례 보고 - (Treatment of Painful Rotator Interval Widening After Subcoracoid Decompression in Elite Archer - Case Report -)

  • 박진영;이승준
    • Clinics in Shoulder and Elbow
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    • 제13권2호
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    • pp.280-285
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    • 2010
  • 목적: 견봉, 오구견봉 인대 및 오구돌기의 끝을 포함하는 오구견봉 간격과 회전근개 사이에서 발생하는 충돌 징후로 인해 발생하는 오구 충돌 증후군은 양궁에서 흔히 볼 수 있는 자세인, 팔을 전방 거상, 내회전 및 내전시에 상완골 소전자가 오구돌기에 접촉하면서 통증이 유발될 수 있다. 대상 및 방법: 보존적 치료로 조절되지 않는 오구 충돌 증후군을 호소한 20세 여자 양궁선수에 대하여 관절경하 오구돌기 성형술 시행 후 발생한 통증을 동반한 회전근 간격 증가에 대하여 관절경하 중첩술을 시행하였다. 결과: 관절경하 회전근 간격 중첩술 시행 후 6개월 추시상, 통증 및 기능 호전을 보이고 성공적으로 양궁 선수로 복귀하였다. 결론: 저자들은 보존적 치료로 조절되지 않는 오구 충돌 증후군을 호소한 20세 여자 양궁선수에 대하여 관절경하 오구돌기 성형술 시행 후 발생한 통증을 동반한 회전근 간격 증가에 대하여 관절경하 중첩술을 시행하여 통증 및 기능 호전을 보이고 성공적으로 양궁 선수로 복귀한 증례를 경험하였기에 보고하고자 한다.

견관절 충돌증후군의 고찰 (Review of Shoulder Joint Impingement Syndrome)

  • 김인섭
    • 대한임상전기생리학회지
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    • 제2권1호
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    • pp.93-100
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    • 2004
  • Shoulder impingement syndrome is cause by the collision of acromion, acrominoclaviclar joint, coracoacromino ligament, coracoid process and synovial sac, biceps brachiialis tendon, rotator cuff muscle. Treatment for Shoulder impingement syndrome is genetally classified into two different methods; preservative method and operational method. Operational method includes rotator cuff sutura, anterior acromioplasty, arthroscope decompression. Preservative method includes rest, medicinal therapy, physical therapy. Physical therapy concentrates on pain control, functional recovery and prevention of disease progress. It is also important for physical therapy to maintain the strength of rotator cuff. Strengthening rotator cuff decreases the collision and helps the stability of shoulder joint. In conclusion, it must be aware that shoulder impingement syndrome and some other shoulder problem demand different treatment, which results in a better outcome.

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회전근개 파열 증후군 (Rotator Cuff Tears Syndrome)

  • 강점덕;김현주
    • 대한정형도수물리치료학회지
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    • 제13권1호
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    • pp.67-72
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    • 2007
  • Anatomy: The rotator cuff comprises four muscles-the subscapularis, the supraspinatus, the infraspinatus and the teres minor-and their musculotendinous attachments. The subscapularis muscle is innervated by the subscapular nerve and originates on the scapula. It inserts on the lesser tuberosity of the humerus. The supraspinatus and infraspinatus are both innervated by the suprascapular nerve, originate in the scapula and insert on the greater tuberosity. The teres minor is innervated by the axillary nerve, originates on the scapula and inserts on the greater tuberosity. The subacromial space lies underneath the acromion, the coracoid process, the acromioclavicular joint and the coracoacromial ligament. A bursa in the subacromial space provides lubrication for the rotator cuff. Etiology: The space between the undersurface of the acromion and the superior aspect of the humeral head is called the impingement interval. This space is normally narrow and is maximally narrow when the arm is abducted. Any condition that further narrows this space can cause impingement. Impingement can result from extrinsic compression or from loss of competency of the rotator cuff. Syndrome: Neer divided impingement syndrome into three stages. Stage I involves edema and/or hemorrhage. This stage generally occurs in patients less than 25 years of age and is frequently associated with an overuse injury. Generally, at this stage the syndrome is reversible. Stage II is more advanced and tends to occur in patients 25 to 40 years of age. The pathologic changes that are now evident show fibrosis as well as irreversible tendon changes. Stage III generally occurs in patients over 50 years of age and frequently involves a tendon rupture or tear. Stage III is largely a process of attrition and the culmination of fibrosis and tendinosis that have been present for many years. Treatment: In patients with stage I impingement, conservative treatment is often sufficient. Conservative treatment involves resting and stopping the offending activity. It may also involve prolonged physical therapy. Sport and job modifications may be beneficial. Nonsteroidal anti - inflammatory drugs(NSAIDS) and ice treatments can relieve pain. Ice packs applied for 20 minutes three times a day may help. A sling is never used, because adhesive capsulitis can result from immobilization.

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