• 한국정보통신학회논문지
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• 제6권7호
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• pp.1064-1068
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• 2002

본 논문에서는 SC-CNN을 이용한 하이퍼카오스 회로에서의 동기화와 비밀통신의 방법을 제시한다. 하이 퍼카오스 회로는 츄아회로의 변형인 N-Double Scroll CNN 회로를 이용하였으며, 동일한 2 개 또는 4개의 셀을 가진 n-double scroll 회로를 이용하여 송 .수신부를 구성하고 이 송 수신부 사이에 구동 동기를 이용한 동기화를 이루었으며, 복원신호에서 상태 변수 $$\chi$_3$ 에 의한 비밀통신이 상태 변수 ${$\chi$_2}, {$\chi$_1}$ 에 의한 방법 보다 우수함을 보였다.

• Journal of information and communication convergence engineering
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• 제1권4호
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• pp.223-228
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• 2003

In this paper, we introduce a hyper-chaos secure communication method using hyper-chaos circuit onsist of State-Controlled Cellular Neural Network SC-CNN). We make a hyper-chaos circuit using SC-CNN with the n-double scroll or Chua's oscillator. A hyper-chaos circuit is created by applying identical n-double scroll or non-identical n-double scroll and Chua's oscillator with weak coupled method to each cell. Hyper-chaos ynchronization was achieved using GS (Generalized Synchronization) method between the transmitter and receiver about each state variable in the SC-CNN. In order to secure communication, we have synthesizing the desired information with a hyper-chaos circuit by adding the information signal to the hyper-chaos signal using the SC-CNN in the transmitter. And then, transmitting the synthesized signal to the ideal channel, we confirm secure communication by separating the information signal and the hyper-chaos signal in the receiver.

• The Korean Journal of Physiology and Pharmacology
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• 제2권3호
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• pp.297-305
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• 1998

This study used in vivo intracellular recording in rat hippocampus to evaluate the effect of lidocaine and MK-801 on the membrane properties and the synaptic responses of individual neurons to electrical stimulation of the commissural pathway. Cells in control group typically fired in a tonic discharge mode with an average firing frequency of $2.4{\pm}0.9$ Hz. Neuron in MK-801 treated group (0.2 mg/kg, i.p.) had an average input resistance of $3.28{\pm}5.7\;M{\Omega}$ and a membrane time constant of $7.4{\pm}1.8$ ms. These neurons exhibited $2.4{\pm}0.2$ ms spike durations, which were similar to the average spike duration recorded in the neurons of the control group. Slightly less than half of these neurons were firing spontaneously with an average discharge rate of $2.4{\pm}1.1$ Hz. The average peak amplitude of the AHP following the spikes in these groups was $7.4{\pm}0.6$ mV with respect to the resting membrane potential. Cells in MK-801 and lidocaine treated group (5 mg/kg, i.c.v.) had an average input resistance of $3.45{\pm}6.0\;M{\Omega}$ and an average time constant of $8.0{\pm}1.4$ ms. The cells were firing spontaneously at an average discharge rate of $0.6{\pm}0.4$ Hz. Upon depolarization of the membrane by 0.8 nA for 400 ms, all of the tested cells exhibited accommodation of spike discharge. The most common synaptic response contained an EPSP followed by early-IPSP and late-IPSP. Analysis of the voltage dependence revealed that the early-IPSP and late-IPSP were putative $Cl^--and\;K^+-dependent$, respectively. Systemic injection of the NMDA receptor blocker, MK-801, did not block synaptic responses to the stimulation of the commissural pathway. No significant modifications of EPSP, early-IPSP, or late-IPSP components were detected in the MK-801 and/or lidocaine treated group. These results suggest that MK-801 and lidocaine manifest their CNS effects through firing pattern of hippocampal pyramidal cells and neural network pattern by changing the synaptic efficacy and cellular membrane properties.