• Journal of the Korean Society of Safety
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• v.12 no.1
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• pp.133-139
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• 1997

The noise generated from a treadband mechanism of a tire has been the subject of this research. In particular, the treadband has been treated as an infinite tensioned beam resting on an elastic foundation which includes damping. The main objective is to predict the sound power generated from a system mentioned above by locating harmonic point forces representing the excitation of treadband at the contact patch. It is possible to predict the sound power radiated from this structure by using wavenumber transformation techniques. In order to find out the minimum radiated sound power, All parameters were varied. Thus, this model can be used as a tire design guide for selecting parameters which produces the minimum noise radiation.

• Transactions of the Korean Society of Automotive Engineers
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• v.7 no.8
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• pp.132-142
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• 1999

In this paper, a new formula for primary and secondary thrust of metal belt CVT is proposed considering variation of band tension, block compression and active arc for each of the primary and secondary pulleys. For the secondary thrust, effective friction coefficient is introduced considering the effect of flange deflection. Nondimensional primary and secondary thrust of the metal belt CVT by the new formula agree well with the experimental results except for low torque range, $0\;<\;{\lambda}\;<\;0.2$ at speed ration i = 1.0. The new formula can be used in design of the primary and secondary thrusts control system for the metal belt CVT.

• Journal of Life Science
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• v.31 no.10
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• pp.913-921
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• 2021

Limb-girdle muscular dystrophy (LGMD) which is characterized by progressive muscle weakening of the hip and shoulder shows both dominant and recessive inheritances with many pathogenic genes including TTN. This study performed to identify genetic causes of a male patient with late onset (45 years old) autosomal recessive LGMD and atrial flutter. By application of the whole exome sequencing, we identified bi-allelic variants of TTN gene in the patient. One allele had a single missense variant of [c.24124G>T (p.V8042F)], while the other allele consisted of three missense variants of [c.29222G>C (p.R9741P) + c.67490A>G (p.H22497R) + c.75376C>T (p.R25126C)]. The p.V8042F allele was transmitted from his mother, while the other haplotype allele was putatively transmitted from his father. His two unaffected sons had only the p.R9741P. These variants have been not reported or rarely reported in the public human genome databases (1,000 Genome, gnomAD, and KRGDB). Most variants were located in the highly conserved immunoglobulin or fibronectin domains and were predicted to be pathogenic by the in silico analyses. The TTN giant protein plays a key role in muscle assembly, force transmission at the Z-line, and maintenance of resting tension in the I-band. In conclusion, we think that these bi-allelic compound heterozygous mutations may play a role as the genetic causes of the LGMD phenotype.