참고문헌
- Bello MJ, Alonso ME, Aminoso C, et al (2004). Hypermethylation of the DNA repair gene MGMT: association with TP53 G:C to A:T transitions in a series of 469 nervous system tumors. Mutat Res, 554, 23-32. https://doi.org/10.1016/j.mrfmmm.2004.02.011
- Bleeker FE, Atai NA, Lamba S, et al (2010). The prognostic IDH1( R132 ) mutation is associated with reduced NADP+-dependent IDH activity in glioblastoma. Acta Neuropathol, 119, 487-94. https://doi.org/10.1007/s00401-010-0645-6
- Butowski NA, Chang SM (2006). Glial tumors: the current state of scientific knowledge. Clin Neurosurg, 53, 106-13.
- Carrillo JA, Lai A, Nghiemphu PL, et al (2012). Relationship between tumor enhancement, edema, IDH1 mutational status, MGMT promoter methylation, and survival in glioblastoma. Am J Neuroradiol, 33, 1349-55. https://doi.org/10.3174/ajnr.A2950
- Dunn GP, Andronesi OC, Cahill DP (2013). From genomics to the clinic: biological and translational insights of mutant IDH1/2 in glioma. Neurosurg Focus, 34, 2.
- Groenendijk FH, Taal W, Dubbink HJ, et al (2011). MGMT promoter hypermethylation is a frequent, early, and consistent event in astrocytoma progression, and not correlated with TP53 mutation. J Neurooncol, 101, 405-17. https://doi.org/10.1007/s11060-010-0274-x
- Hartmann C, Hentschel B, Wick W, et al (2010). Patients with IDH1 wild type anaplastic astrocytomas exhibit worse prognosis than IDH1-mutated glioblastomas, and IDH1 mutation status accounts for the unfavorable prognostic effect of higher age: implications for classification of gliomas. Acta Neuropathol, 120, 707-18. https://doi.org/10.1007/s00401-010-0781-z
- Hegi ME, Diserens AC, Gorlia T, et al (2005). MGMT gene silencing and benefit from temozolomide in glioblastoma. N Engl J Med, 352, 997-1003. https://doi.org/10.1056/NEJMoa043331
- Jesien-Lewandowicz E, Jesionek-Kupnicka D, Zawlik I, et al (2009). High incidence of MGMT promoter methylation in primary glioblastomas without correlation with TP53 gene mutations. Cancer Genet Cytogenet, 188, 77-82. https://doi.org/10.1016/j.cancergencyto.2008.09.015
- Juratli TA, Kirsch M, Geiger K, et al (2012). The prognostic value of IDH mutations and MGMT promoter status in secondary high-grade gliomas. J Neurooncol, 110, 325-33. https://doi.org/10.1007/s11060-012-0977-2
- Koca T, Basaran H, Sezen D, et al (2014). Comparison of linear accelerator and helical tomotherapy plans for glioblastoma multiforme patients. Asian Pac J Cancer Prev, 15, 7811-6. https://doi.org/10.7314/APJCP.2014.15.18.7811
- Lee Y, Scheck AC, Cloughesy TF, et al (2008). Gene expression analysis of glioblastomas identifies the major molecular basis for the prognostic benefit of younger age. BMC Med Genomics, 1, 52. https://doi.org/10.1186/1755-8794-1-52
- Levidou G, El-Habr E, Saetta AA, et al (2010). P53 immunoexpression as a prognostic marker for human astrocytomas: a meta-analysis and review of the literature. J Neurooncol, 100, 363-71. https://doi.org/10.1007/s11060-010-0204-y
- Li S, Jiang T, Li G, Wang Z (2008). Impact of p53 status to response of temozolomide in low MGMT expression glioblastomas: preliminary results. Neurol Res, 30, 567-70. https://doi.org/10.1179/174313208X297913
- Minniti G, Salvati M, Arcella A, et al (2011). Correlation between O6-methylguanine-DNA methyltransferase and survival in elderly patients with glioblastoma treated with radiotherapy plus concomitant and adjuvant temozolomide. J Neurooncol, 102, 311-6. https://doi.org/10.1007/s11060-010-0324-4
- Molenaar RJ, Verbaan D, Lamba S, et al (2014). The combination of IDH1 mutations and MGMT methylation status predicts survival in glioblastoma better than either IDH1 or MGMT alone. Neuro Oncol, 16, 1263-73. https://doi.org/10.1093/neuonc/nou005
- Munoz DM, Guha A (2011). Mouse models to interrogate the implications of the differentiation status in the ontogeny of gliomas. Oncotarget, 2, 590-8. https://doi.org/10.18632/oncotarget.319
- Munoz DM, Tung T, Agnihotri S, et al (2013). Loss of p53 cooperates with K-ras activation to induce glioma formation in a region-independent manner. Glia, 61, 1862-72. https://doi.org/10.1002/glia.22563
- Nagy E, Gajjar KB, Patel, II, et al (2014). MGMT promoter hypermethylation and K-RAS, PTEN and TP53 mutations in tamoxifen-exposed and non-exposed endometrial cancer cases. Br J Cancer, 110, 2874-80. https://doi.org/10.1038/bjc.2014.263
- Nobusawa S, Watanabe T, Kleihues P, Ohgaki H (2009). IDH1 mutations as molecular signature and predictive factor of secondary glioblastomas. Clin Cancer Res, 15, 6002-7. https://doi.org/10.1158/1078-0432.CCR-09-0715
- Noushmehr H, Weisenberger DJ, Diefes K, et al (2010). Identification of a CpG island methylator phenotype that defines a distinct subgroup of glioma. Cancer Cell, 17, 510-22. https://doi.org/10.1016/j.ccr.2010.03.017
- Ohgaki H, Kleihues P (2007). Genetic pathways to primary and secondary glioblastoma. Am J Pathol, 170, 1445-53. https://doi.org/10.2353/ajpath.2007.070011
- Ohgaki H, Kleihues P (2009). Genetic alterations and signaling pathways in the evolution of gliomas. Cancer Sci, 100, 2235-41. https://doi.org/10.1111/j.1349-7006.2009.01308.x
- Olson RA, Brastianos PK, Palma DA (2011). Prognostic and predictive value of epigenetic silencing of MGMT in patients with high grade gliomas: a systematic review and meta-analysis. J Neurooncol, 105, 325-35. https://doi.org/10.1007/s11060-011-0594-5
- Parsons DW, Jones S, Zhang X, et al (2008). An integrated genomic analysis of human glioblastoma multiforme. Science, 321, 1807-12. https://doi.org/10.1126/science.1164382
- Pashaki AS, Hamed EA, Mohamadian K, et al (2014). Efficacy of high dose radiotherapy in post-operative treatment of glioblastoma multiform-a single institution report. Asian Pac J Cancer Prev, 15, 2793-6. https://doi.org/10.7314/APJCP.2014.15.6.2793
- Phillips HS, Kharbanda S, Chen R, et al (2006). Molecular subclasses of high-grade glioma predict prognosis, delineate a pattern of disease progression, and resemble stages in neurogenesis. Cancer Cell, 9, 157-73. https://doi.org/10.1016/j.ccr.2006.02.019
- Sanson M, Marie Y, Paris S, et al (2009). Isocitrate dehydrogenase 1 codon 132 mutation is an important prognostic biomarker in gliomas. J Clin Oncol, 27, 4150-4. https://doi.org/10.1200/JCO.2009.21.9832
- Sarmiento JM, Nuno M, Ortega A, et al (2014). Cystic glioblastoma: an evaluation of IDH1 status and prognosis. Neurosurgery, 74, 71-5: discussion 5-6. https://doi.org/10.1227/NEU.0000000000000200
- Sathornsumetee S, Rich JN, Reardon DA (2007). Diagnosis and treatment of high-grade astrocytoma. Neurol Clin, 25, 1111-39. https://doi.org/10.1016/j.ncl.2007.07.004
- SongTao Q, Lei Y, Si G, et al (2012). IDH mutations predict longer survival and response to temozolomide in secondary glioblastoma. Cancer Sci, 103, 269-73. https://doi.org/10.1111/j.1349-7006.2011.02134.x
- Soussi T, Wiman KG (2007). Shaping genetic alterations in human cancer: the p53 mutation paradigm. Cancer Cell, 12, 303-12. https://doi.org/10.1016/j.ccr.2007.10.001
- Stancheva G, Goranova T, Laleva M, et al (2014). IDH1/IDH2 but not TP53 mutations predict prognosis in Bulgarian glioblastoma patients. Biomed Res Int, 2014, 654727.
- Stander M, Peraud A, Leroch B, Kreth FW (2004). Prognostic impact of TP53 mutation status for adult patients with supratentorial World Health Organization Grade II astrocytoma or oligoastrocytoma: a long-term analysis. Cancer, 101, 1028-35. https://doi.org/10.1002/cncr.20432
- Stupp R, Hegi ME, Mason WP, et al (2009). Effects of radiotherapy with concomitant and adjuvant temozolomide versus radiotherapy alone on survival in glioblastoma in a randomised phase III study: 5-year analysis of the EORTC-NCIC trial. Lancet Oncol, 10, 459-66. https://doi.org/10.1016/S1470-2045(09)70025-7
- Su Y, Yin L, Liu R, et al (2014). Promoter methylation status of MGMT, hMSH2, and hMLH1 and its relationship to corresponding protein expression and TP53 mutations in human esophageal squamous cell carcinoma. Med Oncol, 31, 784. https://doi.org/10.1007/s12032-013-0784-4
- Tabatabai G, Stupp R, van den Bent MJ, et al (2010). Molecular diagnostics of gliomas: the clinical perspective. Acta Neuropathol, 120, 585-92. https://doi.org/10.1007/s00401-010-0750-6
- Turcan S, Rohle D, Goenka A, et al (2012). IDH1 mutation is sufficient to establish the glioma hypermethylator phenotype. Nature, 483, 479-83. https://doi.org/10.1038/nature10866
- van den Bent MJ, Gravendeel LA, Gorlia T, et al (2011). A hypermethylated phenotype is a better predictor of survival than MGMT methylation in anaplastic oligodendroglial brain tumors: a report from EORTC study 26951. Clin Cancer Res, 17, 7148-55. https://doi.org/10.1158/1078-0432.CCR-11-1274
- Verhaak RG, Hoadley KA, Purdom E, et al (2010). Integrated genomic analysis identifies clinically relevant subtypes of glioblastoma characterized by abnormalities in PDGFRA, IDH1, EGFR, and NF1. Cancer Cell, 17, 98-110. https://doi.org/10.1016/j.ccr.2009.12.020
- Weller M, Felsberg J, Hartmann C, et al (2009). Molecular predictors of progression-free and overall survival in patients with newly diagnosed glioblastoma: a prospective translational study of the German Glioma Network. J Clin Oncol, 27, 5743-50. https://doi.org/10.1200/JCO.2009.23.0805
- Wick W, Meisner C, Hentschel B, et al (2013). Prognostic or predictive value of MGMT promoter methylation in gliomas depends on IDH1 mutation. Neurology, 81, 1515-22. https://doi.org/10.1212/WNL.0b013e3182a95680
- Yan W, Zhang W, You G, et al (2012). Correlation of IDH1 mutation with clinicopathologic factors and prognosis in primary glioblastoma: a report of 118 patients from China. PLoS One, 7, 30339. https://doi.org/10.1371/journal.pone.0030339
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