DOI QR코드

DOI QR Code

Furosemide induced medullary nephrocalcinosis mimicking Bartter syndrome

바터씨 증후군으로 오인된 furosemide로 인한 신수질 석회화

  • Kim, Sohee (Department of Internal Medicine, Yousei University College of Medicine) ;
  • Kyung, Chanhee (Department of Internal Medicine, Yousei University College of Medicine) ;
  • Kim, Yong Hoon (Department of Internal Medicine, Yousei University College of Medicine) ;
  • Cho, Jang Ho (Department of Internal Medicine, Yousei University College of Medicine) ;
  • Hwang, Changhyeok (Department of Internal Medicine, Yousei University College of Medicine) ;
  • Lee, Jung Eun (Department of Internal Medicine, Yousei University College of Medicine)
  • 김소희 (연세대학교 의과대학 내과학교실) ;
  • 경찬희 (연세대학교 의과대학 내과학교실) ;
  • 김용훈 (연세대학교 의과대학 내과학교실) ;
  • 조장호 (연세대학교 의과대학 내과학교실) ;
  • 황창혁 (연세대학교 의과대학 내과학교실) ;
  • 이정은 (연세대학교 의과대학 내과학교실)
  • Received : 2013.06.29
  • Accepted : 2013.08.26
  • Published : 2014.06.30

Abstract

Clinical presentation of Bartter syndrome is similar to surrepitious vomiting or use of diuretics. Therefore, precise differential diagnosis of Bartter syndrome is crucial. We report a case of medullary nephrocalcinosis (MNC) induced by furosemide mimicking Bartter syndrome. A 55-year-old female patient visited our hospital with renal dysfunction on basis of hypokalemia and metabolic alkalosis. She had no history of hypertension or drug use except allopurinol and atorvastatin. She did not complain of nausea or vomiting on presentation and the serum magnesium level was normal. We performed ultrasonography, that showed MNC. For these reasons, we suspected Bartter syndrome and corrected the electrolyte imbalance. During outpatient follow up, we found that the patient had been taking 400 mg of furosemide daily for 30 years. We could diagnose furosemide induced MNC, and recommended to her to reduce the amount of furosemide.

Keywords