References
- Akirav EM, Preston-Hurlburt P, Garyu J, et al (2012). RAGE expression in human T cells: a link between environmental factors and adaptive immune responses. PLoS One, 7, e34698. https://doi.org/10.1371/journal.pone.0034698
- Barqasho B, Nowak P, Abdurahman S, et al (2010). Implications of the release of high-mobility group box 1 protein from dying cells during human immunodeficiency virus type 1 infection in vitro. J Gen Virol, 91, 1800-9. https://doi.org/10.1099/vir.0.016915-0
- Beyer C, Stearns NA, Giessl A, et al (2012). The extracellular release of DNA and HMGB1 from Jurkat T cells during in vitro necrotic cell death. Innate Immun, 18, 727-37. https://doi.org/10.1177/1753425912437981
- Bianchi ME, Agresti A (2005). HMG proteins: dynamic players in gene regulation and differentiation. Curr Opin Genet Dev, 15, 496-506. https://doi.org/10.1016/j.gde.2005.08.007
- Boxus M, Twizere JC, Legros S, et al (2008). The HTLV-1 Tax interactome. Retrovirology, 5, 76. https://doi.org/10.1186/1742-4690-5-76
- Bustin M (1999). Regulation of DNA-dependent activities by the functional motifs of the high-mobility-group chromosomal proteins. Mol Cell Biol, 19, 5237-46.
- Christian Grant, Michael Nonnemacher, Pooja Jain, et al (2006). CCAAT/enhancer-binding proteins modulate human T cell leukemia virus type 1 long terminal repeat activation. Virology, 348, 354-69. https://doi.org/10.1016/j.virol.2005.12.024
- Easley R, Carpio L, Guendel I, et al (2010). Human T-Lymphotropic Virus Type 1 Transcription and Chromatin-Remodeling Complexes. J Virol, 84, 4755-68. https://doi.org/10.1128/JVI.00851-09
- Gerlitz G, Hock R, Ueda T, et al (2009). The dynamics of HMG protein-chromatin interactions in living cells, Biochem Cell Biol, 87, 127-37. https://doi.org/10.1139/O08-110
- Goncalves DU, Proietti FA, Ribas JG, et al (2010). Epidemiology, treatment, and prevention of human T-cell leukemia virus type 1-associated diseases. Clin Microbiol Rev, 23, 577-89. https://doi.org/10.1128/CMR.00063-09
-
He Q, You H, Li XM, et al (2012). HMGB1 promotes the synthesis of pro-IL-
$1{\beta}$ and pro-IL-18 by activation of p38 MAPK and NF-${\kappa}B$ through receptors for advanced glycation end-products in macrophages. Asian Pac J Cancer Prev, 13, 1365-70. https://doi.org/10.7314/APJCP.2012.13.4.1365 - Jube S, Rivera ZS, Bianchi ME, et al (2012). Cancer cell secretion of the DAMP protein HMGB1 supports progression in malignant mesothelioma. Cancer Res, 72, 3290-301. https://doi.org/10.1158/0008-5472.CAN-11-3481
- Kang R, Tang D, Schapiro NE, et al (2013). The HMGB1/RAGE inflammatory pathway promotes pancreatic tumor growth by regulating mitochondrial bioenergetics. Oncogene, doi: 10.1038/onc.2012.631.
- Kannian P, Green PL (2010). Human T lymphotropic virus type 1 (HTLV-1): molecular biology and oncogenesis. Viruses, 2, 2037-77. https://doi.org/10.3390/v2092037
- Kostova N, Zlateva S, Ugrinova I, et al (2010). The expression of HMGB1 protein and its receptor RAGE in human malignant tumors. Mol Cell Biochem, 337, 251-8. https://doi.org/10.1007/s11010-009-0305-0
- Kwon H, Ogle L, Benitez B, et al (2005). Lethal cutaneous disease in transgenic mice conditionally expressing type I human T cell leukemia virus Tax. J Biol Chem, 280, 35713-22. https://doi.org/10.1074/jbc.M504848200
-
Lavorgna A, Harhaj EW (2012). An RNA interference screen identifies the Deubiquitinase STAMBPL1 as a critical regulator of human T-cell leukemia virus type 1 tax nuclear export and NF-
${\kappa}B$ activation. J Virol, 86, 3357-69. https://doi.org/10.1128/JVI.06456-11 - Lee H, Park M, Shin N, et al (2012). High mobility group box-1 is phosphorylated by protein kinase C zeta and secreted in colon cancer cells. Biochem Biophys Res Commun, 424, 321-6. https://doi.org/10.1016/j.bbrc.2012.06.116
- Liu F, Zhang Y, Peng Z, et al (2012). High expression of high mobility group box 1 (hmgb1) predicts poor prognosis for hepatocellular carcinoma after curative hepatectomy. J Transl Med, 10, 135. https://doi.org/10.1186/1479-5876-10-135
- Luo Y, Chihara Y, Fujimoto K, et al (2013). High mobility group box 1 released from necrotic cells enhances regrowth and metastasis of cancer cells that have survived chemotherapy. Eur J Cancer, 49, 741-51. https://doi.org/10.1016/j.ejca.2012.09.016
- Mohammad G, Siddiquei MM, Othman A, et al (2013). High mobility group box-1 protein activates inflammatory signaling pathway components and disrupts retinal vascular-barrier in the diabetic retina. Exp Eye Res, 107, 101-9. https://doi.org/10.1016/j.exer.2012.12.009
- Moisy D, Avilov SV, Jacob Y, et al (2012). HMGB1 protein binds to influenza virus nucleoprotein and promotes viral replication. J Virol, 86, 9122-33. https://doi.org/10.1128/JVI.00789-12
- Nowak P, Abdurahman S, Lindkvist A, Troseid M, Sönnerborg A (2012). Impact of HMGB1/TLR Ligand Complexes on HIV-1 Replication: Possible Role for Flagellin during HIV-1 Infection. Int J Microbiol, 2012, 263836.
- Ohmori H, Luo Y, Kuniyasu H (2011). Non-histone nuclear factor HMGB1 as a therapeutic target in colorectal cancer. Expert Opin Ther Targets, 15, 183-93. https://doi.org/10.1517/14728222.2011.546785
-
Patrick WP Ng, Hidekatsu Iha, Yoichi Iwanaga, et al (2001). Genome-wide expression changes induced by HTLV-1 Tax: evidence for MLK-3 mixed lineage kinase involvement in Tax-mediated NF-
${\kappa}B$ activation. Oncogene, 20, 4484-96. https://doi.org/10.1038/sj.onc.1204513 - Polakowski N, Han H, Lemasson I (2011). Direct inhibition of RNAse T2 expression by the HTLV-1 Viral protein Tax. Viruses, 3, 1485-500. https://doi.org/10.3390/v3081485
- Stoetzer OJ, Fersching DM, Salat C, et al (2013). Circulating immunogenic cell death biomarkers HMGB1 and RAGE in breast cancer patients during neoadjuvant chemotherapy. Tumour Biol, 34, 81-90. https://doi.org/10.1007/s13277-012-0513-1
- Tahaei SM, Mohebbi SR, Fatemi SR, et al (2011). Low frequency of human T-cell lymphotropic virus 1 antibodies in Iranian gastric cancer patients in comparison to controls. Asian Pac J Cancer Prev, 12, 2447-50.
- Troseid M, Sonnerborg A, Nowak P (2011). High mobility group box protein-1 in HIV-1 infection. Curr HIV Res, 9, 6-10. https://doi.org/10.2174/157016211794582632
- Uchiyama T, Yodoi J, Sagawa K, et al (1977). Adult T-cell leukemia: clinical and hematologic features of 16 cases. Blood, 50, 481-92.
- Vidal AU, Gessain A, Yoshida M, et al (1994). Molecular epidemiology of HTLV type I in Japan: evidence for two distinct ancestral lineages with a particular geographical distribution. AIDS Res Hum Retroviruses, 10, 1557-66. https://doi.org/10.1089/aid.1994.10.1557
- Mao XJ, Wang GF, Chen ZJ, et al (2012). Expression of HMGB1 and its Clinical Significance in T-cell Lymphoma. Asian Pac J Cancer Prev, 13, 5569-71. https://doi.org/10.7314/APJCP.2012.13.11.5569
- Yoshida M, Miyoshi I, Hinuma Y (1982). Isolation and characterization of retrovirus from cell lines of human adult T-cell leukemia and its implication in the disease. Proc Natl Acad Sci USA, 79, 2031-5. https://doi.org/10.1073/pnas.79.6.2031
- Yu Y, Xie M, Kang R, et al (2012). HMGB1 is a therapeutic target for leukemia. Am J Blood Res, 2, 36-43.
Cited by
- High Mobility Group B Proteins, Their Partners, and Other Redox Sensors in Ovarian and Prostate Cancer vol.2016, pp.1942-0994, 2016, https://doi.org/10.1155/2016/5845061
- Abundant expression of HMGB1 in human T-cell lymphotropic virus type I-infected T-cell lines and high plasma levels of HMGB1 in patients with adult T-cell leukemia vol.7, pp.4, 2014, https://doi.org/10.3892/ol.2014.1851