References
- Sims, G. P., D. C. Rowe, S. T. Rietdijk, R. Herbst, and A. J. Coyle. 2010. HMGB1 and RAGE in inflammation and cancer. Annu. Rev. Immunol. 28: 367-388. https://doi.org/10.1146/annurev.immunol.021908.132603
- Lotze, M. T. and K. J. Tracey. 2005. High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal. Nat. Rev. Immunol. 5: 331-342. https://doi.org/10.1038/nri1594
- Degryse, B., T. Bonaldi, P. Scaffidi, S. Müller, M. Resnati, F. Sanvito, G. Arrigoni, and M. E. Bianchi. 2001. The high mobility group (HMG) boxes of the nuclear protein HMG1 induce chemotaxis and cytoskeleton reorganization in rat smooth muscle cells. J. Cell Biol. 152: 1197-1206. https://doi.org/10.1083/jcb.152.6.1197
- Rouhiainen, A., J. Kuja-Panula, E. Wilkman, J. Pakkanen, J. Stenfors, R. K. Tuominen, M. Lepäntalo, O. Carpén, J. Parkkinen, and H. Rauvala. 2004. Regulation of monocyte migration by amphoterin (HMGB1). Blood 104: 1174-1182. https://doi.org/10.1182/blood-2003-10-3536
- Sappington, P. L., R. Yang, H. Yang, K. J. Tracey, R. L. Delude, and M. P. Fink. 2002. HMGB1 B box increases the permeability of Caco-2 enterocytic monolayers and impairs intestinal barrier function in mice. Gastroenterology 123:790-802. https://doi.org/10.1053/gast.2002.35391
- Andersson, U., H, Wang, K. Palmblad, A. C. Aveberger, O. Bloom, H. Erlandsson-Harris, A. Janson, R. Kokkola, M. Zhang, H. Yang, and K. J. Tracey. 2000. High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes. J. Exp. Med. 192: 565-570. https://doi.org/10.1084/jem.192.4.565
- Youn, J. H., Y. J. Oh, E. S. Kim, J. E. Choi, and J. S. Shin. 2008. High mobility group box 1 protein binding to lipopolysaccharide facilitates transfer of lipopolysaccharide to CD14 and enhances lipopolysaccharide-mediated TNF-alpha production in human monocytes. J. Immunol. 180: 5067-5074. https://doi.org/10.4049/jimmunol.180.7.5067
- Sha, Y., J. Zmijewski, Z. Xu, and E. Abraham. 2008. HMGB1 develops enhanced proinflammatory activity by binding to cytokines. J. Immunol. 180: 2531-2537. https://doi.org/10.4049/jimmunol.180.4.2531
- Bonaldi, T., F. Talamo, P. Scaffidi, D. Ferrera, A. Porto, A. Bachi, A. Rubartelli, A. Agresti, and M. E. Bianchi. 2003. Monocytic cells hyperacetylate chromatin protein HMGB1 to redirect it towards secretion. EMBO J. 22: 5551-5560. https://doi.org/10.1093/emboj/cdg516
- Kim, J. H., S. J. Kim, I. S. Lee, M. S. Lee, S. Uematsu, S. Akira, and K. I. Oh. 2009. Bacterial endotoxin induces the release of high mobility group box 1 via the IFN-beta signaling pathway. J. Immunol. 182: 2458-2466. https://doi.org/10.4049/jimmunol.0801364
- Zhang, X., D. Wheeler, Y. Tang, L. Guo, R. A. Shapiro, T. J. Ribar, A. R. Means, T. R. Billiar, D. C. Angus, and M. R. Rosengart. 2008. Calcium/calmodulin-dependent protein kinase (CaMK) IV mediates nucleocytoplasmic shuttling and release of HMGB1 during lipopolysaccharide stimulation of macrophages. J. Immunol. 181: 5015-5023. https://doi.org/10.4049/jimmunol.181.7.5015
- Wang, L., I. Tassiulas, K. H. Park-Min, A. C. Reid, H. Gil-Henn, J. Schlessinger, R. Baron, J. J. Zhang, and L. B. Ivashkiv. 2008. 'Tuning' of type I interferon-induced Jak- STAT1 signaling by calcium-dependent kinases in macrophages. Nat. Immunol. 9: 186-193.
- Tokumitsu, H., H. Inuzuka, Y. Ishikawa, M. Ikeda, I. Saji, and R. Kobayashi. 2002. STO-609, a specific inhibitor of the Ca(2+)/calmodulin-dependent protein kinase kinase. J. Biol. Chem. 277: 15813-15818. https://doi.org/10.1074/jbc.M201075200
- Karaghiosoff, M., R. Steinborn, P. Kovarik, G. Kriegshäuser, M. Baccarini, B. Donabauer, U. Reichart, T. Kolbe, C. Bogdan, T. Leanderson, D. Levy, T. Decker, and M. Müller. 2003. Central role for type I interferons and Tyk2 in lipopolysaccharide- induced endotoxin shock. Nat. Immunol. 4:471-477. https://doi.org/10.1038/ni910
- Weighardt, H., S. Kaiser-Moore, S. Schlautkötter, T. Rossmann-Bloeck, U. Schleicher, C. Bogdan, and B. Holzmann. 2006. Type I IFN modulates host defense and late hyperinflammation in septic peritonitis. J. Immunol. 177:5623-5630. https://doi.org/10.4049/jimmunol.177.8.5623
- Liu, X., M. Yao, N. Li, C. Wang, Y. Zheng, and X. Cao. 2008. CaMKII promotes TLR-triggered proinflammatory cytokine and type I interferon production by directly binding and activating TAK1 and IRF3 in macrophages. Blood 112: 4961-4970. https://doi.org/10.1182/blood-2008-03-144022
Cited by
- Extracellular High-Mobility Group Box 1 Protein (HMGB1) as a Mediator of Persistent Pain vol.20, pp.None, 2014, https://doi.org/10.2119/molmed.2014.00176
- Release of Neuronal HMGB1 by Ethanol through Decreased HDAC Activity Activates Brain Neuroimmune Signaling vol.9, pp.2, 2012, https://doi.org/10.1371/journal.pone.0087915
- Stress-altered synaptic plasticity and DAMP signaling in the hippocampus-PFC axis; elucidating the significance of IGF-1/IGF-1R/CaMKIIα expression in neural changes associated with a prolonged e vol.353, pp.None, 2012, https://doi.org/10.1016/j.neuroscience.2017.04.008
- High Mobility Group Box-1 (HMGb1): Current Wisdom and Advancement as a Potential Drug Target : Miniperspective vol.61, pp.12, 2012, https://doi.org/10.1021/acs.jmedchem.7b01136
- LPS Induces Active HMGB1 Release From Hepatocytes Into Exosomes Through the Coordinated Activities of TLR4 and Caspase-11/GSDMD Signaling vol.11, pp.None, 2012, https://doi.org/10.3389/fimmu.2020.00229
- Signaling pathways and intervention therapies in sepsis vol.6, pp.1, 2012, https://doi.org/10.1038/s41392-021-00816-9