References
- Herschman HR. Primary response genes induced by growth factors and tumor promoters.AnnuRevBiochem 1991;60: 281-319 https://doi.org/10.1146/annurev.bi.60.070191.001433
- WilliamsCS,MannM,DuBoisRN. Theroleofcyclooxy genases in inflammation, cancer, and development. Oncogene 1999;18:7908-7916 https://doi.org/10.1038/sj.onc.1203286
- TuckerON,Dannenberg AJ, Yang EK, et al. Cyclooxygenase-2 expression is up-regulated in human pancreatic cancer. Cancer Res 1999;59:987-990
- Uefuji K, Ichikura T, Mochizuki H, Shinomiya N. Expression of cyclooxygenase-2 protein in gastric adenocarcinoma. J Surg Oncol 1998;69:168-172 https://doi.org/10.1002/(SICI)1096-9098(199811)69:3<168::AID-JSO9>3.0.CO;2-0
- Sano H, Kawahito Y, Wilder RL, et al. Expression of cyclooxygenase-1 and 2 in human colorectal cancer. Cacner Res 1995;55:3785-3789
- Battu S,Rigaud M, Beneytout JL. Resistance to apoptosis and cyclooxygenase-2 expression in a human adenocarcinoma celllineHT29CL.19A.AnticancerRes1998;18:3579-3583
- ChanG.BoyleJO,YangEK,etal. Cyclooxygenase-2expression is up-regulated in squamous cell carcinoma of the head and neck. Cancer Res 1999;59:991-994
- Elder DJE, Halton DE, Hague A, Paraskeva C. Induction of apoptotic cell death in human colorectal carcinoma cell lines by a cyclooxygenase-2 (COX-2)- selective nonsteroidal anti-inflammatory drug: independence from COX-2 protein expression. Clin Cancer Res 1997;3:1679-1683
- Hida T, Yatabe Y, Achiwa H, et al. Increased expression of cyclooxygenase 2 occurs frequently in human lung cancers,specifically in adenocarcinomas.CancerRes1998; 58:3761-3764
- Milas L, Kishi K, Hunter N, Mason K, Masferrer JL, Tofilon PJ. Enhancement of tumor response to -radiation by an inhibitor of cyclooxygenase-2 enzyme. J Natl Cancer Inst 1999;91:1501-1504 https://doi.org/10.1093/jnci/91.17.1501
- Taketo MM. Cyclooxygenase-2 inhibitors in tumorigenesis (part II). J Natl Cancer Inst 1998;90:1609-1620 https://doi.org/10.1093/jnci/90.21.1609
- Rioux N, Castonguay A. Prevention of NNK-induced lung tumorigenesis in A/J mice by acetylsalicylic acid and NS- 398. Cancer Res 1998;58:5354-5360
- Kalgutkar AS, Crews BC, Rowlinson SW, Garner C, Seibert K, MarnettLJ. Aspirin-like moleculesthatcovalently inactivate cyclooxygenase-2. Science 1998;280:1268- 1270 https://doi.org/10.1126/science.280.5367.1268
- CrewTE,ElderDJE,Paraskeva CA. Cyclooxygenase-2(COX-2) selective non-steroidal anti-inflammatory drug enhances the growth inhibitory effect of butyrate in colorectal carcinoma cells expressing COX-2 protein: regulation of COX-2 by butyrate. Carcinogenesis 2000;21:69-77 https://doi.org/10.1093/carcin/21.1.69
- Hsu AL, Ching TT,WangDS,SongX,RangnekarVM, Chen CS. The cyclooxygenase-2 inhibitorCelecoxib induces apoptosis by blocking Akt activation in human prostate cancer cells independentlyofBcl- 2.JBiolChem2000;275: 11397-11403
- LiuXH,YaoS,KirschenbaumA,LevineAC. NS398, a selective cyclooxygenase-2 inhibitor, induces apoptosis and down-regulates bcl-2 expression in LNCaP cells. Cancer Res 1998;58:4245-4249
- Hara A, Yoshimi N, Niwa M, Ino N, Mori H. Apoptosis induced by NS-398, a selective cyclooxygenase-2 inhibitor, in human colorectal cancer cell lines. Jpn J. Cancer Res 1997;88:600-604
- Sheng H, ShaoJ,KirklandSC,etal. Inhibitionofhumancolon cancer cell growth by selective inhibition of cyclooxygenase- 2. J Clin Invest 1997;99:2254-2259 https://doi.org/10.1172/JCI119400
-
Sheng H, Shao J, Morrow JD, Beauchamp RD, DuBois RN. Modulation of apoptosis and Bcl-2 expression by prostaglandin E
$_2$ in human colon cancer cells. Cancer Res 1998; 58:362-366 - MilasL,FurutaY,HunterN,NishiguchiI,RunkelS. Dependence of indomethacin-induced potentiation of murine tumor radioresponse on tumor host immunocompetence. Cancer Res 1990;50:4473-4477
- Palayoor ST, Bump EA, Calderwood SK, Bartol S, Coleman CN. Combined antitumor effect of radiation and ibuprofen in human prostate carcinoma cells. Clin Cancer Res 1998;4:763-771
- Furuta Y, Hunter N, Barkley T Jr, Hall E, Milas L. Increase in radioresponse of murine tumors by treatment with indomethacin. Cancer Res 1988;48:3008-3013
- Kishi K, Petersen S, Petersen C, et al. Preferential enhancement of tumor radioresponse by a cyclooxygenase- 2 inhibitor. CancerRes2000;60:1326-1331
- Petersen C, Petersen S, Milas L, Lang FF, Tofilon PJ. Enhancement of intrinsic tumor cell radiosensitivity induced by a selective cyclooxygenase-2 inhibitor. Clin Cancer Res 2000;6:2513-2520
- Pyo H, Choy H, Amorino GP, et al. A selective cyclooxygenase- 2 inhibitor, NS-398, enhances the effect of radiation in vitro and in vivo preferentially on the cells which express cyclooxygenase-2. Clin Cancer Res 2001;7:2998-3005
- DuBois RN, Shao J, Tsujii M, Sheng H, BeauchampRD. G1 delay in cells overexpressing prostaglandin endoperoxide synthase-2. Cancer Res 1996;56: 733-737
- Toyoshima T, Kamijo R, Takizawa K, SumitaniK,ItoD,Nagumo M. Inhibitor of cyclooxygenase-2 induces cell-cyclearrest in the epithelial cancer celllineviaup-regulation of cyclin dependent kinase inhibitor p21. Br J Cancer 2002; 86:1150-1156 https://doi.org/10.1038/sj.bjc.6600183
- Furuta Y, Hall ER, Sanduja S, Barkley T Jr, Milas L. Prostaglandin production by murine tumors as a predictor for therapeutic response to indomethacin. Cancer Res 1988;48: 3002-3007
- Gilroy D, Saunders M,WuK.COX-2expressionandcell cycleprogressioninhumanfibroblasts.AmJPhysiolCellPhysiol 2001;281:C188-C194
- Gilroy D, Saunders M, Sansores-Garcia L, Matijevic- Aleksic N, Wu K. Cell cycle-dependent expression of cyclooxygenase-2 in human fibroblasts. FASEB J 2001;15: 288-90 https://doi.org/10.1096/fj.00-0573fje
- Eric J Hall. Repairof radiation damage andthedose-rate effect. In: Eric J Hall, eds. Radiobiology for the radiobiologist. 5th ed. Philadelphia, PA: Lippincott Williams & Wilkins Co. 2000:67-90
- ChangH,WengC.Cyclooxygenase-2 level andcultureconditions influence NS398-induced apoptosis and caspase ctivation in lung cancer cells. Oncol Rep 2001;8:1321-1325