A Study on the Defence Effect of Banhasasim-tang for White Rat's Acute Duodenal Injury

흰쥐의 급성 십이지장 손상에 대한 반하사심탕의 방어효과에 관한 연구

  • 한이수 (동의대학교 한의과대학 내과학교실) ;
  • 최준혁 (동의대학교 한의과대학 내과학교실) ;
  • 임성우 (동의대학교 한의과대학 내과학교실)
  • Published : 2002.09.01

Abstract

Objectives : Banhasasim-tang has been clinically used to treat upper gastric intestinal discomfort. The object of this study is to examine the defense effect of Banhasasim-tang for acute duodenal injury of the mouse. Methods and Materials : Twenty-one rats were divided into 3 groups and treated as follows: the control group was untreated mice. The ADE group was acute duodenal-damage-elicited mice. The BST group was Banhasasim-tang treated mice before acute duodenal damage elicitation. The groups were examined with common morphology, paneth cells in intestinal crypt, absorptive cells and goblet cells in epithelium, cell division in mucose, COX-l as mucosal protector, COX-2 (which appears to play an important role in inflammation), IL-2R-inducing cellular immuno-chainreaction, and the distribution of apoptotic cells. Results : 1. Common morphology: the ADE group was observed with duodenal injury - loss of villi, infiltration of cells concerned to inflammation (lymphocytes, granular leukocytes) to submucosal layer - by hemorrhagic erosions, while the BST group was seen the same as normal in proportion to increasing treatment time before injury. 2. Histochemical change: the ADE group was observed with noticeable decreased distribution of absorptive cells with microvilli, acid mucin secreted goblet cell, neutral mucin secreted goblet cell, paneth cells compared to the normal group. The BST group was seen to have distribution of epithelium cells resembling normal in proportion to increasing treatment time before injury. 3. Imnunohistochemical change: the ADE group showed a change of factors leading to duodenal injury as reduce of cytokinesis, COX-1, increase of COX-2, IL-2R-. In contrast, the BST group tended to reduction of cytokinesis, COX-1, increase of COX-2, IL-2R- in proportion to increasing taking time before injury. 4. Apoptosis change: the ADE group showed increasing apoptosis cells, in contrast to the BST group which was the same as normal in proportion to increasing treatment time before injury. Conclusions : According to the above results, by increasing the defense system of mucosal epithelium, Banhasasim-tang is thought to effectively protect tissue against ulcers resulting from acute duodenal injury.

Keywords

References

  1. Gastrointestinal Disease.(2nd ed.) Sturdevant. Duodenal ulcer Richard AL.;Sleisenger MH(et al.)
  2. N Engl J Med. v.331 Bleeding peptic ulcer. Laine L;Peter W.
  3. 脾胃病證治精要 李家庚;傳延齡
  4. 實用中西醫結合診斷治療學 陳貴延;楊恩澍
  5. 경희한의대논문집 v.9 수점산이 위궤양 및 진통에 미치는 영향. 김영준;류기원
  6. 경희한의대논문집 v.8 단삼보혈탕 및 보화환이 위궤양에 미치는 영향. 박동원
  7. 대한한의학회지 v.19 no.1 가미향사육군자탕이 Indometacin 유도 위점막손상에 미치는 항산화 효과. 김경선;신흥묵
  8. 內經類證 素伯未
  9. 仲景全書 張仲景
  10. Bockus’s gastroenterology (3rd ed.) Etiology and pathology of peptic ulcer. Shay H;Sun DCH.
  11. Langenbeck's Arch. v.386 Incidence and pathophysiology of peptic ulcer bleeding. Georg D, Arlt;Maarkus Leyh.
  12. 소화기학 서울대학교 의과대학(편)
  13. 응용약물학회지 v.2 위장질환 치료용 의약조성물(DWP301)의 일반약리작용. 임승욱(외 12인)
  14. 基礎藥理學實驗 久保田和彦(外 3人)
  15. 대한소화기학회지 v.34 no.5 소화성 궤양 환자에서 Helicobacterpylori박멸 전후의 전정부 위염 양상의 변화. 김유선(외 10인)
  16. 대한소화기내시경학회지 v.19 no.2 출혈성 소화성 궤양의 장기 재발율. 박의련(외 7인)
  17. 대한한방내과학회지 v.20 no.2 四君子湯合蒼朮地楡湯이 위점막손상에 미치는 영향. 손정숙;임성우
  18. 대한한방내과학회지 v.22 no.2 桃花湯이 백성의 소화성 궤양 및 장관수송능에 미치는 영항. 이익행(외 4인)
  19. 대한한의학회지 v.19 no.1 괴화산이 항궤양효과 및 혈액응옥작용에 미치는 실험적 연구. 강채춘(외 2인)
  20. 대한한의학회지 v.18 no.1 대건중탕의 항궤양 및 위장관에 미치는 효과에 관한 실험적 연구. 김혁규;백태현
  21. 반하사심탕,생강사심탕 및 감초사심탕의 효능에 관한 실험적 비교연구. 이진섭
  22. Am J med. v.105 Gastrointestinal side effects of nonsteroidal anti-inflammatory drug. Cryer B;Kimmey MB.
  23. Inflamm res. v.47 Interleukin-15 and its role in chronic inflammatory diseases. Kirman I:Vainer B;Nielsen OH.
  24. Transplant Proceed. v.32 Upregulated intragraft gene expression, ICAM-1 and IL-2R molecules, and apoptotic epithelial cells during regection of rat small intestine allgrafts. Li YX;Li N;Li YS;Wu B;Li JS.
  25. J Gastroenterol. v.33 Roles of COX-1 and COX-2 in gastrointestinal pathophysiology. Sakamoto C.
  26. Gastroenterology. v.98 NSAID-induced gastropathy : a neutrophil dependent process. Wallace JL;Keenan LM;Granger DN.
  27. Ann Intern Med. v.115 Risk for serious gastrointestinal complications related to use of nonsteroidal antiinflammatory drugs. Gabriel SE;Jaakkimainen L;Bombardier C.
  28. Free Radical Bio Med. v.26 Apoptosisprocess in mokey snall intestinal epithelium: 2. possible role of oxidative stress. Madesh M;Benad O;Balasubramanian KA.
  29. Inflamm res. v.49 Cyclooxygenase-2 - its rich diversity of roles and possible application of its selective inhibitors. Katori M;Majima M.
  30. Life Sciences. v.61 Effects of selective cyclooxygenase-2 inhibitors on alkaline secretory and mucosal ulcergenic responses in rat duodenum. Hirata T;Ukawa H;Kitamura M;Takeuchi K.
  31. Abodm Imaging. v.23 NSAlD injury to the small intestine. Zalev AH;Gardiner GW;Warren RE.